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JEFFREY MAASSEN, MD
MD
Ophthalmology Physician
NPI: 1124065883IndividualAccepts Medicare
Specialties, Licenses & Credentials
Ophthalmology PhysicianPrimary
Ophthalmology
Code: 207W00000X
36554(IA)
Education
UNIVERSITY OF IOWA, RJ & L CARVER COLLEGE OF MEDICINE
Class of 2002
Research & Publications (20)
The nuclear appearance of ERK1/2 and p38 determines the sequential induction of ATF2-Thr71 and ATF2-Thr69 phosphorylation by serum in JNK-deficient cells.
PMID 19647037·Mol Cell Endocrinol·2009
7-preclinical
Genetic association analysis of 13 nuclear-encoded mitochondrial candidate genes with type II diabetes mellitus: the DAMAGE study.
PMID 19209188·Eur J Hum Genet·2009
4-observational
Phosphorus-31 magnetic resonance spectroscopy of skeletal muscle in maternally inherited diabetes and deafness A3243G mitochondrial mutation carriers.
PMID 19097109·J Magn Reson Imaging·2009
8-other
Neuropeptide Y polymorphism significantly magnifies diabetes and cardiovascular disease risk in obesity: the Hoorn Study.
PMID 18043699·Eur J Clin Nutr·2009
8-other
Combined effects of single-nucleotide polymorphisms in GCK, GCKR, G6PC2 and MTNR1B on fasting plasma glucose and type 2 diabetes risk.
PMID 19533084·Diabetologia·2009
8-other
Fatty acid-induced mitochondrial uncoupling in adipocytes as a key protective factor against insulin resistance and beta cell dysfunction: do adipocytes consume sufficient amounts of oxygen to oxidise fatty acids?
PMID 18317722·Diabetologia·2008
8-other
Loss of 50% of excess weight using a very low energy diet improves insulin-stimulated glucose disposal and skeletal muscle insulin signalling in obese insulin-treated type 2 diabetic patients.
PMID 18080107·Diabetologia·2008
3-trial
Variants of CDKAL1 and IGF2BP2 affect first-phase insulin secretion during hyperglycaemic clamps.
PMID 18618095·Diabetologia·2008
8-other
Fatty acid-induced mitochondrial uncoupling in adipocytes as a key protective factor against insulin resistance and beta cell dysfunction: a new concept in the pathogenesis of obesity-associated type 2 diabetes mellitus.
PMID 17712547·Diabetologia·2007
8-other
The A3243G tRNALeu(UUR) mutation induces mitochondrial dysfunction and variable disease expression without dominant negative acting translational defects in complex IV subunits at UUR codons.
PMID 17656376·Hum Mol Genet·2007
8-other
Lessons that can be learned from patients with diabetogenic mutations in mitochondrial DNA: implications for common type 2 diabetes.
PMID 18089949·Curr Opin Clin Nutr Metab Care·2007
6-review
New insights in the molecular pathogenesis of the maternally inherited diabetes and deafness syndrome.
PMID 16632100·Endocrinol Metab Clin North Am·2006
6-review
Mitochondrial dysfunction in adipocytes: the culprit in type 2 diabetes?
PMID 16491392·Diabetologia·2006
7-preclinical
The role of c-Jun N-terminal kinase, p38, and extracellular signal-regulated kinase in insulin-induced Thr69 and Thr71 phosphorylation of activating transcription factor 2.
PMID 16601071·Mol Endocrinol·2006
7-preclinical
Insulin-mediated phosphorylation of the proline-rich Akt substrate PRAS40 is impaired in insulin target tissues of high-fat diet-fed rats.
PMID 17130464·Diabetes·2006
7-preclinical
Sustained activation of the mammalian target of rapamycin nutrient sensing pathway is associated with hepatic insulin resistance, but not with steatosis, in mice.
PMID 17006666·Diabetologia·2006
7-preclinical
Diabetes-associated mitochondrial DNA mutation A3243G impairs cellular metabolic pathways necessary for beta cell function.
PMID 16736129·Diabetologia·2006
8-other
Mitochondrial diabetes and its lessons for common Type 2 diabetes.
PMID 17052206·Biochem Soc Trans·2006
8-other
The HADHSC gene encoding short-chain L-3-hydroxyacyl-CoA dehydrogenase (SCHAD) and type 2 diabetes susceptibility: the DAMAGE study.
PMID 17065362·Diabetes·2006
4-observational
The p38 mitogen-activated protein kinase inhibitor SB203580 reduces glucose turnover by the glucose transporter-4 of 3T3-L1 adipocytes in the insulin-stimulated state.
PMID 15665038·Endocrinology·2005
7-preclinical
Data courtesy of the U.S. National Library of Medicine (NLM). Ltrl is not affiliated with or endorsed by NLM.
Contact & Hours
- Address
- 1650 1ST AVE NE
CEDAR RAPIDS, IA 52402 - Phone
- (319) 362-3937
Quick Facts
- NPI
- 1124065883
- Entity Type
- Individual
- Gender
- Male
- Medicare
- Accepted
- Specialties
- 1
- Locations
- 1
- Years in Practice
- 24
- Publications
- 20
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